Skip to main content
I'm still waiting for the magic moment that will tell me that biologists are using cell surface antigens to reprogram cancer cells.

However, there was news recently about how scientists are closely examining histones as a means towards reprogramming. You see, histones are used by the cell to keep DNA in check. As any of you who have had the misfortune of having to unravel a ball of string know, stringy material like DNA would be impossible to keep orderly if there weren't some means of keeping them neatly spooled. This is where histones come in. They are the spools for DNA. It seems as if there might be a mechanism in place-involving histones--whereby the histones are manipulated by epigenetic factors in order to facilitate or defacilitate the transcription of DNA.

In other words, Cancer cells are causing certain histones to allow a certain DNA expression. If we can shut down the active histones, we may be able to shut down cancer; but this would only work if histones were found to be specific enough for each type of cancer. If you closed down the histones from a metastatic lung cell, you do not want your treatment to affect any other histone (as for example, a normal liver cell's histones).

To summarize previous posts to this blog, cells, like ants, people, and organizations of all kinds need to communicate with their neighbors. When they fail at this, cells try communicating using language that previously worked for them. But that language was used at an embryonic stage and no one else in the adult organism is using that language. Cancer cells then become outcasts and only succeed in creating chaotic embryos that never develop because no other cell is speaking quite the same language. They end up metastasizing to a certain area because there they think they have found a home.

Comments

Popular posts from this blog

This article was recently published in PLOS. This is a great scientific publishing medium that doesn't charge anyone a cent to read their stuff, unlike Science and Nature and the others that hoard their publications at the expense of humanity. Anyway, the article points out that a boy being treated with stem cells for a rare genetic condition developed tumors of the brain and spinal cord. These results should come as no surprise for readers of this blog. The cells injected into the nerve tissues were allografts--they were obtained from a source other than the patients own tissues. These clearly will not be at home in another person's body and they will begin to express embryonic Cell Recognition Factors in the hope that they could establish communication. As we have learned here, a cell must know exactly where it is. Such communication must exist. If it does not, it would mean that A) any manner of invader could easily make its home in another multicellular organism, or B) any...

What about the unattached cell?

While this procedure is not what I had in mind when I spoke of being able to reprogram the cancer cell, I'm nevertheless glad that reprogramming was found to be possible for the unattached cancer cell. The unattached immune cell is needed everywhere in the body and it would make sense for a cancer derived from an immune cell to differentiate into other immune cells as needed. This is what was found by the Stanford researchers--reprogramming of leukemia cells into normal granulocytes and macrophages via supplied ligands or transcription factors. The initial observation from Stanford came about as a result of a shotgun approach. This approach should also be used for the solid tumor and if their cocktail doesn't do it, I would consider extracting Embryonic Cell Surface molecules from various developmental time points. Finding the right CRF for the differentiation of  solid tumors is right around the corner. My next post will be one showing what the CRF theory helps explain wh...

Biological Pathways Symbology